A., & Weiskrantz, L. (1999). Attention without awareness in blindsight. Proceedings of the Royal Society of London,
Series B, 266, 1805-1811; Kentridge, R. W., Heywood, C. A., & Weiskrantz, L. (2004). Spatial attention speeds discrimination without awareness in blindsight. Neuropsychologia, 42, 831-835.] demonstrated just such a dissociation in the blindsight subject GY. Here, we test whether the dissociation generalizes to the normal population. We presented observers with pairs of coloured discs, each masked by the subsequent presentation of a coloured annulus. The discs acted as primes, speeding discrimination of the colour of the annulus when they matched in colour and slowing it when they differed. We show that learn more the location of attention modulated the size of this priming effect. Cediranib chemical structure However, the primes were rendered invisible by metacontrast-masking and remained unseen despite being attended. Visual attention could therefore facilitate processing of an invisible target and cannot, therefore, be a sufficient precondition for visual awareness. (c) 2007 Elsevier Ltd. All rights reserved.”
“Gammaherpesvirus infection is associated with an increased incidence of lymphoproliferative disease in immunocompromised hosts. Murine gammaherpesvirus 68 (gamma HV68) infection of BALB beta(2)-microglobulin-deficient
(BALB beta(2)m(-/-)) mice provides an animal model for analysis of the mechanisms responsible for the induction of a lymphoproliferative disease, atypical lymphoid hyperplasia (ALH), that is pathologically similar to posttransplant lymphoproliferative disease associated with Epstein-Barr virus infection. Here we report that the gamma HV68 v-cyclin and v-bcl-2 genes are required for the efficient induction gamma HV68-associated ALH in BALB beta(2)m(-/-) mice, while the v-GPCR gene is dispensable for ALH induction.
In contrast to these findings, deletion of the viral M1 gene enhanced ALH. Thus, gamma HV68 genes can either inhibit or enhance the induction of lymphoproliferative disease in immunocompromised mice.”
“Whereas research on blindsight customarily defines the correct responses to all visual stimuli presented to the cortically blind field, we here introduced a small number see more of unexpected ‘no stimulus’ trials in a localization task, to discover whether they would elicit the same responses as blind field targets. As no correct responses existed for the blank stimuli, our subjects, three hemianopic and one normal monkey, and one human hemianope who was aware of many blind-field targets, could either respond to these catch trials as to a target or refrain from responding. Visual stimuli were presented singly at four possible positions, two in the blind field of the hemianopes, and all subjects correctly localized the vast majority of targets in either hemifield.